Nursing Shortage in the United States

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Critical Issue Proposal

Recruitment of Nurses from the Philippines due to the Nursing Shortage in the United States

Our local and national health care systems are facing a nursing shortage of mass proportions. We are now only beginning to understand the loss of necessary talent in the health care industry, specifically nurses, and the impact that such a deficit will have on the quality of health care across the United Sates. The problem is simple-there are not enough nurses to fill our current actual health care needs; there is expected to be an even greater gap as our needs increase. The nurses are aging and fewer people are training in the nursing profession. The nurses are unhappy with many aspects of their career choice. Their workloads are heavy across the board, the hours are long and tiring, they have no support staff, mandatory overtime after an already exhausting 1 hour shift; and like in all areas inadequate pay. All of these are factors that I will give support to in order to describe why its so necessary to bring nurses here from the Philippines. The Philippines have a trained nursing workforce willing to come to the United States for the betterment of themselves, their families and to help us where there is a cry for help.

At the same time the nurses are retiring, the baby boomers are reaching their 60s. The need for the nurses is at an all time high. From my readings of essays and articles so far, it seems that everything is going to hit at once - nursing retirements and the boomers needing the care simultaneously. The bottom line is that the nursing school system is falling behind the current nursing demand by at least % and increasing year over year.

The shortage is very real and very different from any other nursing shortage in the past. Before when there was a shortage, there was a backlog of students ready to graduate and to join the work force, but that is not the case anymore. There is an all time low of nurses entering the workforce; there is a shortage of nurses with adequate education to meet patient needs; there are changes in the health care environment as it once was. Health care is becoming based around making money, not taking care of the patients, which is an entirely different topic. The regulations are at an all time high, which is bringing on major debt for the hospitals, homes for the aging and private care institutions; the need is in every state in the United States and at every level of nursing.

I find it fascinating that our United States of America has to reach out to other countries to help us with something so serious as a nursing shortage. Why have we waited so long to solve this problem and once again have to go to a foreign country for help instead of stepping up to the plate and revising our nursing practices here to have our own people fill these positions. The shortage is not new; its just extremely severe.

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Of Mice and Men

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In John Steinbecks novel, Of Mice and Men, the lead character, George, and his companion, Lennie, spend the majority of the novel working on a ranch. Lennie, who is quite slow, carries out several actions that go against Georges mind-set. Lennies continuous uncouth behavior eventually places him in a dangerous dilemma. After Lennie commits a crime, George has to make a decision that could end in the death of his friend. Many factors contribute to Georges final decision to kill Lennie; George knew that the men would kill Lennie if he did not, and he felt that he should do it as quickly and painlessly as possible. He also did not want the men to think that he was part of the crime that Lennie committed, and did not want to have to deal with Lennie for the rest of his life if he had helped Lennie escape.

Georges first reasoning for killing Lennie was that he knew that the men on the ranch would kill Lennie if they found him alive. Lennie committed a horrendous crime. Not only did Lennie kill a human being, but he killed Curleys wife. The men on the ranch were outraged that Lennie would kill another man, let alone Curleys wife. Curley was known to have a bad temper; he tended to pick fights with the other men. One of the workers on the ranch describes Curley as a man who is, "alla time picking scraps…seems like Curley aint givin nobody a chance" (Steinbeck, ). The men knew that Curley would be outraged, and even the civil-minded Slim knew that Curley would stop at nothing to kill Lennie. He said, "Curleys gonna want to shoot 'im. Curleys still mad about his hand" (Steinbeck, 106). And they were right. Curley was furious that Lennie would do such a thing. He proclaimed, "I know who done it…that big son-of-a-bitch done it. I know he done it. Why-ever body else was out there playin horseshoes…Im gonna get him. Im going for my shotgun. Ill kill the son-of-a-bitch myself. Ill shoot 'im in the guts" (Steinbeck, 105-106). Curley was so determined to kill Lennie and George knew that we would succeed; none of the men would stop Curley. George knew that if the men found Lennie, they would most likely make him suffer and ruthlessly murder him. George was trying to protect Lennie from this horrific ordeal.

Another reason why George decided to kill Lennie was that he didnt want the other men to think that he was in on the killing of Curleys wife. George and Candy knew that Curleys wife was dead before the other men discovered her. George became scared when he realized that if the other men walked in on himself and Candy standing over Curleys wifes dead body, they would assume the worst- that they killed Curleys wife. George didnt want the other men to think he was in on her killing, so he told Candy, "Now you listen. The guys might think I was in on it. Im gonna go in the bunk house. Then in a minute you come out and tell the guys about her, and Ill come along and make like I never seen her. Will you do that? So the guys wont think I was in on it?" (Steinbeck, 104). He took every precaution to make sure that they wouldnt suspect him. Later, when the men are searching for Lennie, Curley screamed at him, "You George! You stick with us so we dont think you had nothin to do with this" (Steinbeck, 108). George followed along, obeying Curley so they wouldnt blame him for Curleys wifes death. Once he found Lennie, he had to kill him before the men came to find Lennie alive. If they found George and Lennie alive together, the men would suspect George of trying to help Lennie escape. Buy Of Mice and Men term paper

George also didnt want his hopes and dreams to be tied down by Lennie. He knew that he had to take care of Lennie, even though he didnt want to sometimes. He told Lennie

"Whatever we aint got, thats what you want. God amighty, if I was alone I could live so easy. I could go get a job an work, an no trouble. No mess at all, and when the end of the month come I could take my fifty bucks and go into town and get whatever I want…An whatta I got…I got you! You cant keep a job and you lose me ever job I get. Jus keep me shovin all over the country all the time. An that aint the worst. You get in trouble. You do bad things and I got to get you out…You crazy son-of-a-bitch. You keep me in hot water all the time" (Steinbeck, 11-1).

George knew that life without Lennie would be much easier and would give him freedom. He wanted to be able to live his own life, without having to look after Lennie. Keeping him out of trouble was close to impossible, and taking care of Lennie became a chore that George didnt want to deal with anymore.

There were many ideas that George had to think about while deciding whether or not to kill Lennie. But eventually he came to realize that he didnt want the men to be cruel to Lennie, he didnt want the men to think he was involved in Curleys wifes murder, and he came to understand that he didnt want to have to take care of Lennie anymore. All of these factors forced George to realize that killing Lennie himself was the best choice at the time.

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Diabetes and Eating Disorders

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Insulin-dependent diabetes mellitus (IDDM) is one of the most common chronic illnesses of childhood and adolescence in North America.1 Although most young patients with IDDM are healthy, up to 40 percent eventually have diabetes-related microvascular complications., The risk is greater in those whose diabetes is poorly controlled.4 Eating behavior is categorized under three commonly selected categories.4 Highly disordered eating is defined as the occurrence of one or more of the following forms of disordered behavior at least twice per week binge eating, omission or under dosing of insulin to promote weight loss, self-induced vomiting, or use of laxatives.4 Moderately disordered eating is defined as the occurrence of one or more of these forms of disordered behavior at least twice per month, but less than twice per week.4 Nondisordered eating is defined as the absence of disordered behavior or its occurrence less than twice per month.4 Up to one third of young women with IDDM have eating disturbances,5 which may affect the management of diabetes. Treatment of type 1 diabetes involves constant monitoring of food intake. In addition, the good glycemic control necessary to reduce the risk of long-term complications is associated with weight gain.6 In young women, these two factors, along with individual, family and social factors, can lead to an increased incidence of eating disorders, which can disrupt glycemic control and increase the risk of long-term complications.6,7,8 The coexistence of eating disorders and diabetes is associated with non-cooperation with treatment for diabetes,7 omission or under dosing of insulin to induce glycosuria and promote weight loss,8 and impaired metabolic control; 8 however, long-term effects of disordered eating on complications of diabetes are not known. Nevertheless, it is still determined that disordered eating behavior is associated with microvascular complications in young women with IDDM.

From June to December 188 (base line), 11 girls and women 1 to 18 years old were invited to participate in a self-reported survey of eating attitudes and behavior.7 These girls and women had previously diagnosed IDDM and were being followed in the diabetes clinic of the Hospital for Sick Children in Toronto. This represented all girls and women in this age group who attended the clinic during this period, except for one patient with cerebral palsy. Between July 1 and January 14 (follow-up), all of the participants were contacted again. Approximately one third were still attending the diabetes clinic, and the remainder had been referred to an adult treatment setting. Between study entry and follow-up, the research group did not have any contact with the study participants, except that one provided medical care for some of the patients at the clinic and another saw several patients for psychiatric assessment. The treatment for patients at the diabetes clinic between study entry and follow-up included regular quarterly visits and IDDM management in a multidisciplinary setting. It was also clinical practice to recommend a psychosocial assessment for patients with persistently high hemoglobin A1c levels or disturbed eating attitudes and behavior. During the follow-up interval, twenty-one patients reported that they had been assessed or treated for one or more of the following an eating disorder (), depression (), family problems () and other mental problems (4).

At base line and follow-up, demographic and clinical information was collected, height and weight were measured and body-mass index was calculated. Self-reported episodes of ketoacidosis and severe hypoglycemia in the preceding year were documented. A behavior related to eating and weight self-administered questionnaire was given at base line and follow-up. This questionnaire, which obtains information about eating habits from the previous three months, was changed to include diabetes-related items, including omission or under dosing insulin to promote weight loss. The patients either completed the questionnaire during their clinical visits or completed it at home and returned it by mail. Even with reminder calls, some questionnaires (eight at base line and nine at follow-up) were not returned.

Hemoglobin A1c was measured at base line and follow-up. Also, the urinary albumin excretion rate, a predictor of diabetic neuropathy, was determined at follow-up in both 1 and 4 hour urine samples; however, the results from the 4-hour samples were used as the most reliable measurement. Microalbuminuria was defined as an albumin excretion rate of at least 15 but less than 00 nanograms per minute, and macroalbuminuria as a rate of at least 00 nanograms per minute. Diabetic retinopathy was detected at follow-up by a retinal specialist who did not know the persons eating habits, hemoglobin A1c levels, or urinary albumin excretion rate. The level of retinopathy was derived by giving a greater weight to the eye with the higher level.10 With this classification, level 10 indicates no diabetic retinopathy; level 0, very mild retinopathy; level 0, mild nonproliferative retinopathy; levels 40 to 55 moderate-to-severe nonproliferative retinopathy; and level 60 or higher, mild to high risk proliferative retinopathy.10 Custom writing service can write essays on Diabetes and Eating Disorders

The results then showed that 107 (88 percent) of the 11 eligible girls and women participated at base line because 8 did not return their questionnaires and 6 refused to participate. Then, 1 (85 percent) of these 107 girls participated at follow-up because did not return their questionnaires and 5 could not be located. The characteristics of the patients at base line and follow-up are shown in Table 1.11

Also, the 16 patients who participated at base line but not at follow-up did not differ from the 1 who completed both assessments, in terms of age, age at onset of diabetes, duration of diabetes, hemoglobin A1c values, BMI and eating status at base line. Among the 11 patients who refused to participate or failed to return their questionnaires at follow-up, were classified as having highly disordered eating and had moderately disordered eating. The prevalence and persistence of disordered eating behavior are shown in Table .

Intentional omission or underdosing of insulin and dieting for weight loss increased in frequency from base line to follow-up. Binge eating, self-induced vomiting, and dieting for weight loss tended to continue at follow-up if they were not present at base line. Also, at base line, of the 1 young women met the criteria for highly disordered eating, 17 met the criteria for moderately disordered eating and 65 met the criteria for nondisordered eating. The nine patients with highly disordered eating did not differ from the others in age, but in duration of diabetes (+/-4 vs. 6+/-4 years). Table also suggested that disordered-eating status tended to persist over time because of the 6 patients with highly or moderately disordered eating at base line, 16 remained in these categories and 10 improved. Of the 65 patients with nondisordered eating at base line, 14 had disordered eating at follow-up.

At base line, the patients with highly disordered eating had a substantially higher hemoglobin A1c value than those with moderately disordered eating and nondisordered eating. Among the 14 patients who had constant disordered eating behavior and whose hemoglobin A1c values were measured at follow-up, the values were similarly high at both assessments. In the nine patients whose eating status improved, so did their hemoglobin A1c and decreased from .7+/-. to 7.6+/-1.4 percent. 71 of the 1 women had ophthalmologic examinations at follow-up and 4 of the 71 were found to have some degree of retinopathy. Sixteen had mild retinopathy, eight patients had nonproliferative retinopathy and one had advanced preproliferative retinopathy in one eye and early proliferative in the other. Urinary albumin excretion was measured in 7 of the 1 patients at follow-up. Twelve had microalbumuria (range, 15 to 66 nanograms per minute) and three had macroalbumuria (range, to 47 nanograms per minute). This association between disordered-eating status at base line and diabetes-related microvascular complications at follow up is shown in Table .

Results show that retinopathy was more common in patients with disordered eating at base line, but abnormal urinary albumin excretion was not.

Therefore, the most striking finding of this study is that some degree of retinopathy was present at follow-up in more than 85% of young women with IDDM who had highly disordered eating at base line, as compared with 4% in those with moderately disordered eating. Only 4% of those, however, with nondisordered eating had some degree of retinopathy. Furthermore, disordered-eating status accounted for more of the explained variance in a model predicting retinopathy than did duration of diabetes, an established risk factor for microvascular complications.4 Also, the average length of diabetes in the patients may not have been long enough for neuropathy to occur in enough patients to suspect an association with disordered-eating status. The limitations of the study, however, include incomplete participation in the follow-up medical examinations, limited reliability on the honesty of the self-report assessment, and absence of base line evaluations of microvascular complications.

This study, therefore, confirmed that in young women with diabetes, eating disorders do persist, to increase in frequency throughout adolescence, and to be predictors of poor metabolic control and their related complications. The increased prevalence of behavior designed to promote weight loss at follow-up is not surprising because more of the patients had reached the aged where eating disorders were at a higher risk. Also, apart from dieting to lose weight, intentional omission or under dosing of insulin was the most common means of inducing weight loss.

Prevention and early treatment of eating disorders in young women are important to prevent long-term complications and mortality. The health risk of these conditions is increased when they are associated with diabetes because of their effect on metabolic control and metabolic complications. In diabetic women with diabetes, the increased focus on eating and the weight gain associated with good glycemic control likely increase their susceptibility to abnormal eating. Although there is an emphasis on keeping weight down, a healthy diet should be stressed. Good nutritional counseling to help patients avoid weight gain and family counseling to improve communication between patients and their families may help decrease the risk. Intentional insulin omission is a frequent means of preventing weight gain or increasing weight loss in adolescent females with type 1 diabetes. Eating disorders should be suspected in patients with recurrent ketoacidosis or poor glycemic control that is resistant to attempts at improvement. Treatment includes decreasing dietary restraint, promoting healthy eating and either psychiatric counseling or psychologic intervention, or both. It is always important to remember that an eating disorder can be hidden easily and for many years, usually until the consequences are irreversible. 1.Drash AL. The epidemiology of insulin-dependent diabetes mellitus. Clin Invest Med 187; 10 4-46.

.Krolewski AS, Warram JH, Christlieb AR, Busick EJ, Kahn CR. The changing natural history of nephropathy in type I diabetes. Am J Med 185; 78785-74.

.Krolewski AS, Warram JH, Rand LI, Christlieb AR, Busick EJ, Kahn CR. Risk of proliferative diabetic retinopathy in juvenile-onset type I diabetes a 40-yr follow-up study. Diabetes Care 186;44-45.

4.The Diabetes Control and Complications Trial Research Group. The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. N Engl J Med 1;77-86.

5.Rodin GM, Johnson LE, Garfinkel PE, Daneman D, Kenshole AB. Eating disorders in female adolescents with insulin dependent diabetes mellitus. Int J Psychiatry Med 186;164-57.

6.The Diabetes Control and Complications Trial Research Group. The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. N Engl J Med 1;(14)77-86

7.Steel JM, Young RJ, Lloyd GG, Clarke BF. Clinically apparent eating disorders in young diabetic women associations with painful neuropathy and other complications. BMJ 187;485-86.

8.Rodin G, Craven J, Littlefield C, Murray M, Daneman D. Eating disorders and intentional insulin undertreatment in adolescent females with diabetes. Psychosomatics 11; 171-176.

.McKenna MJ, Arias C, Feldkamp CS, Whitehouse FW. Microalbuminuria in clinical practice. Arch Intern Med 11; 1511745-1747.

10.Diabetic Retinopathy Study Research Group. Report 7 a modification of the Airlie House classification of diabetic retinopathy. Invest Ophthmol Vis Sci 181;110-6.

11.Cole TJ, Freeman JV, Preece MA. Body mass index reference curves for the UK, 10. Arch Dis Child 15;75-.

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